Would it be appropriate to develop a vaccine against B. fragilis or P. gingivalis? Why or why not? If not, what would be the most effective types of prevention? Why might P. aeruginosa produce so many...

Would it be appropriate to develop a vaccine against B. fragilis or P. gingivalis? Why or why not? If not, what would be the most effective types of prevention?

Why might P. aeruginosa produce so many virulence factors? Why is cell-to-cell signaling, which controls the production of many of these factors, so important? Could these signaling systems become a target for antibiotics?