The immunologic mechanisms involved in acute poststreptococcal glomerulonephritis include
a. tubular blocking by precipitates of bacteria and antibody reactions.
b. deposition of immune complexes and complement along the GBM.
c. thickening of the GBM from autoimmune microangiopathic changes.
d. destruction of glomeruli by proteolytic enzymes contained in the GBM.
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