A SO-year-old male patient of he patic cirrhosis with ascitis and pedal edema was treated with tab Furosemide 80 mg twice a day, in addition to bed rest, suitable dietary advice and vitamin supplementation. He started passing la rger quantity of urine and the ascitis/edema started regressing. After a week, he was brought with incoherent talking, drowsiness, tremor and ataxia. The relatives informed that for the past 2 days he was no longer passing the increased amount of urine as at the start of medication. Serum K• measurement found a value of 2.8 mEq/L. (a) What is the cause of the neurological symptoms and diminution of the diuretic response to fu rosemid e? Was the choice of the diuretic appropriate? (b) How should this patient be managed at the present stage?
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